Night Hag

sleep paralysis and nightmares can converge because both originate in REM sleep physiology, a state marked by vivid dream imagery, heightened limbic activation, and skeletal muscle atonia. During sleep paralysis, REM-related muscle inhibition persists into wakefulness while cortical awareness returns, and in some cases dream mentation, particularly threat-related or dysphoric content, continues intruding into conscious perception. This can produce intense hallucinations such as sensed presence, chest pressure, suffocation, or looming figures that feel like a continuation of a preceding nightmare. Neurobiologically, this reflects simultaneous activation of REM dream-generating networks including the amygdala alongside partial restoration of wakeful awareness, while motor output remains suppressed. The convergence is more likely under conditions that destabilize REM regulation such as sleep deprivation, stress, trauma exposure, or PTSD, resulting in an experience where the boundary between nightmare and waking reality becomes perceptually indistinct.